APE1-mediated DNA damage repair provides survival advantage for esophageal adenocarcinoma cells in response to acidic bile salts-Reference-Cited by-同舟云学术

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APE1-mediated DNA damage repair provides survival advantage for esophageal adenocarcinoma cells in response to acidic bile salts

Published:2016-02-25 Issue:13 Volume:7 Page:16688-16702
ISSN:1949-2553
Container-title:Oncotarget
language:en
Short-container-title:Oncotarget

Author:

Hong Jun¹,Chen Zheng¹,Peng Dunfa¹,Zaika Alexander¹²³,Revetta Frank⁴,Washington M. Kay⁴,Belkhiri Abbes¹,El-Rifai Wael¹²³

Affiliation:

1. Department of Surgery, Vanderbilt University Medical Center, Nashville, Tennessee, USA

2. Department of Cancer Biology, Vanderbilt University Medical Center, Nashville, Tennessee, USA

3. Department of Veterans Affairs, Tennessee Valley Healthcare System, Nashville, Tennessee, USA

4. Department of Pathology, Vanderbilt University Medical Center, Nashville, Tennessee, USA

Publisher

Impact Journals, LLC

Subject

Oncology

Cited by 26 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Reflux conditions induce E-cadherin cleavage and EMT via APE1 redox function in oesophageal adenocarcinoma;Gut;2023-09-21

2. Revisiting Two Decades of Research Focused on Targeting APE1 for Cancer Therapy: The Pros and Cons;Cells;2023-07-20

3. Lactobacillus sp. Facilitate the Repair of DNA Damage Caused by Bile-Induced Reactive Oxygen Species in Experimental Models of Gastroesophageal Reflux Disease;Antioxidants;2023-06-21

4. Role of APE1/Ref-1 in hydrogen peroxide-induced apoptosis in human renal HK-2 cells;Kidney Research and Clinical Practice;2023-05-23

5. Protein adduction causes non-mutational inhibition of p53 tumor suppressor;Cell Reports;2023-01

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