Toll-like 4 receptor /NFκB inflammatory/miR-146a pathway contributes to the ART-correlated preterm birth outcome-Reference-Cited by-同舟云学术

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Toll-like 4 receptor /NFκB inflammatory/miR-146a pathway contributes to the ART-correlated preterm birth outcome

Published:2016-09-12 Issue:45 Volume:7 Page:72475-72485
ISSN:1949-2553
Container-title:Oncotarget
language:en
Short-container-title:Oncotarget

Author:

Zhong Xinqi¹,Jiang Yi-Zhou²,Liu Peiwen³,He Wenzhi⁴,Xiong Zhongtang⁵,Chang Weijie¹,Zhu Jiandong¹,Cui Qiliang¹

Affiliation:

1. Department of Pediatrics, The Third Affiliated Hospital of Guangzhou Medical University, Guangdong, China

2. Institute for Advanced Study, Shenzhen University, Shenzhen, Guangdong, China

3. Department of Pathogen Biology, School of Public Health and Tropical Medicine, Southern Medical University, Guangdong, China

4. Experimental Department of Institute of Gynecology and Obstetrics, The Third Affiliated Hospital of Guangzhou Medical University, Guangdong, China

5. Department of Pathology, The Third Affiliated Hospital of Guangzhou Medical University, Guangdong, China

Publisher

Impact Journals, LLC

Subject

Oncology

Cited by 23 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Exosomal miR-146a-5p derived from human umbilical cord mesenchymal stem cells can alleviate antiphospholipid antibody-induced trophoblast injury and placental dysfunction by regulating the TRAF6/NF-κB axis;Journal of Nanobiotechnology;2023-11-13

2. An updated review on cell signaling pathways regulated by candidate miRNAs in coronary artery disease;Non-coding RNA Research;2023-09

3. Role of Transcription Factors in the Management of Preterm Birth: Impact on Future Treatment Strategies;Reproductive Sciences;2022-09-21

4. Association of Polymorphisms in miR146a, an Inflammation-Associated MicroRNA, with the Risk of Idiopathic Recurrent Spontaneous Miscarriage: A Case-Control Study;Disease Markers;2022-04-30

5. Downregulation of miR-21 is Involved in the Pathogenesis of Infection-Induced Preterm Birth by Targeting NF-κB;Reproductive Sciences;2022-03-07

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