Identification of FRA-1 as a novel player in pancreatic cancer in cooperation with a MUC1: ERK signaling axis
Author:
Affiliation:
1. Eppley Institute for Research in Cancer and Allied Diseases, University of Nebraska Medical Center, Omaha, NE 68198, USA
2. University of New Mexico, Albuquerque, NM 87131, USA
Publisher
Impact Journals, LLC
Subject
Oncology
Cited by 11 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Aptamer-mediated nano-therapy for pancreatic cancer;Recent Advances in Nanocarriers for Pancreatic Cancer Therapy;2024
2. Overexpression of FRA1 (FOSL1) Leads to Global Transcriptional Perturbations, Reduced Cellular Adhesion and Altered Cell Cycle Progression;Cells;2023-09-24
3. Altered glycosylation in pancreatic cancer and beyond;Journal of Experimental Medicine;2022-05-06
4. FOSL1 promotes cholangiocarcinoma via transcriptional effectors that could be therapeutically targeted;Journal of Hepatology;2021-08
5. CCR7-CCL21 axis promotes the cervical lymph node metastasis of tongue squamous cell carcinoma by up-regulating MUC1;Journal of Cranio-Maxillofacial Surgery;2021-07
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