Activation of the stress protein response prevents the development of pulmonary edema by inhibiting VEGF cell signaling in a model of lung ischemia‐reperfusion injury in rats-Reference-Cited by-同舟云学术

Activation of the stress protein response prevents the development of pulmonary edema by inhibiting VEGF cell signaling in a model of lung ischemia‐reperfusion injury in rats

Published:2006-06-22 Issue:9 Volume:20 Page:1519-1521
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Godzich M.¹,Hodnett M.¹,Frank J. A.²,Su G.²,Pespeni M.¹,Angel A.¹,Howard M. B.¹,Matthay M. A.²,Pittet J. F.²,Godzich M.³,Hodnett M.³,Frank J. A.²,Su G.²,Pespeni M.³,Angel A.³,Howard M. B.³,Matthay M. A.²,Pittet J. F.²

Affiliation:

1. Laboratory of Surgical Research, Departments of Anesthesia, Surgery, University of California San Francisco California USA

2. Medicine and Cardiovascular Research Institute, University of California San Francisco California USA

3. Laboratory of Surgical ResearchDepartments of Anesthesia, Surgery, University of California San Francisco California USA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.05-4708fje

Reference44 articles.

1. Ischemia–Reperfusion–induced Lung Injury

2. Egr-1, a master switch coordinating upregulation of divergent gene families underlying ischemic stress

3. PKCβ regulates ischemia/reperfusion injury in the lung

4. Vascular endothelial growth factor and related molecules in acute lung injury

5. Lung Overexpression of the Vascular Endothelial Growth Factor Gene Induces Pulmonary Edema

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