Altered gene expression and functions of mitochondria in human nephrotic syndrome

Author:

Holthöfer Harry1,Kretzler Matthias2,Haltia Anni1,Solin Marja-Liisa1,Taanman Jan-Willem3,Schägger Hermann4,Kriz Wilhelm5,Kerjaschki Dontscho6,Schlöndorff Detlef2

Affiliation:

1. The Haartman InstituteDivision of Bacteriology and ImmunologyUniversity of Helsinki Finland

2. Medical PoliclinicUniversity of Munich Munich Germany

3. Department of Clinical NeurosciencesUniversity of London London U.K.

4. University of Frankfurt Frankfurt Germany

5. Department of AnatomyUniversity of Heidelberg Heidelberg Germany

6. Department of PathologyUniversity of Vienna Vienna Austria

Funder

Helsingin Yliopisto

Paulon Säätiö

Päivikki ja Sakari Sohlbergin Säätiö

Academy of Finland

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Reference49 articles.

1. Current status of the structural and functional basis of glomerular filtration and proteinuria;Kanwar Y. S.;Semin. Nephrol.,1991

2. How does glomerular epithelial cell injury contribute to progressive glomerular damage;Rennke H. G.;Kidney Int.,1994

3. Main Features of the Congenital Nephrotic Syndrome

4. Congenital nephrotic syndrome

5. Tissue specificity of rat mitochondrial dimethylglycine dehydrogenase expression

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