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C5a receptor and thymocyte apoptosis in sepsis

  • Published:2002-04-23 Issue:8 Volume:16 Page:887-888
  • ISSN:0892-6638
  • Container-title:The FASEB Journal
  • language:en
  • Short-container-title:FASEB j.

Author:

Riedemann Niels C.1,Guo Ren‐Feng1,Laudes Ines J.1,Keller Katie1,Sarma Vidya J.1,Padgaonkar Vaishalee1,Zetoune Firas S.1,Ward Peter A.1

Affiliation:

1. Department of PathologyUniversity of Michigan Medical School Ann Arbor MI 48109

Funder

National Institutes of Health

National Heart, Lung, and Blood Institute

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Reference36 articles.

1. Protective effects of C5a blockade in sepsis

2. Endotoxin‐induced shock in the rat;Smedegard G.;A role for C5a. Am J Pathol,1989

3. THE DERIVATION OF TWO DISTINCT ANAPHYLATOXIN ACTIVITIES FROM THE THIRD AND FIFTH COMPONENTS OF HUMAN COMPLEMENT

4. Generation of C5‐derived lysosomal enzyme‐releasing activity (C5a) by lysates of leukocyte lysosomes;Goldstein I. M.;J Immunol,1974

5. Oxygen radicals mediate endothelial cell damage by complement-stimulated granulocytes. An in vitro model of immune vascular damage.

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