Melatonin‐induced neuroprotection after closed head injury is associated with increased brain antioxidants and attenuated late‐phase activation of NF‐κB and AP‐1

Author:

Beni Sara M.1,Kohen Ron2,Reiter Russel J.3,Tan Dun‐Xian3,Shohami Esther1

Affiliation:

1. Departments of Pharmacology The Hebrew University of Jerusalem Jerusalem Israel

2. Pharmaceutics School of Pharmacy The Hebrew University of Jerusalem Jerusalem Israel

3. Department of Cellular and Structural Biology The University of Texas San Antonio Texas

Funder

National Science Foundation

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Reference73 articles.

1. The free radical pathology and the microcirculation in the major central nervous system disorders;Demopoulos H. B.;Acta Physiol. Scand. Suppl.,1980

2. The role of oxygen radicals in the pathobiology of traumatic brain injury;Povlishock J. T.;Hum. Cell,1992

3. Reactive Oxygen Radicals in Signaling and Damage in the Ischemic Brain

4. REVIEW ■ : Calcium and the Pathogenesis of Traumatic CNS Injury: Cellular and Molecular Mechanisms

5. Alterations in cerebral energy metabolism induced by traumatic brain injury

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