RAGE‐dependent signaling in microglia contributes to neuroinflammation, Aβ accumulation, and impaired learning/memory in a mouse model of Alzheimer's disease-Reference-Cited by-同舟云学术

RAGE‐dependent signaling in microglia contributes to neuroinflammation, Aβ accumulation, and impaired learning/memory in a mouse model of Alzheimer's disease

Published:2009-11-11 Issue:4 Volume:24 Page:1043-1055
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Fang Fang¹,Lue Lih-Fen²,Yan Shiqiang¹³,Xu Hongwei¹⁴,Luddy John S.¹,Chen Doris¹,Walker Douglas G.²,Stern David M.⁵,Yan Shifang¹,Schmidt Ann Marie¹,Chen John X.⁶,Yan Shirley ShiDu¹⁷⁸

Affiliation:

1. Department of SurgeryCollege of Physicians and SurgeonsColumbia UniversityNew York New York USA

2. Laboratory of NeuroinflammationSun Health Research InstituteSun City Arizona USA

3. College of Chemistry and Chemical EngineeringLangzhou UniversityLangzhou China

4. Department of ImmunologyHarbin Medical UniversityHarbin China 150086

5. College of MedicineUniversity of CincinnatiCincinnati Ohio USA

6. Department of NeurologyMemorial Sloan-Kettering Cancer CenterNew York New York USA

7. Department of PathologyCollege of Physicians and SurgeonsColumbia UniversityNew York New York USA

8. Taub Institute for Research on Alzheimer's Disease and the Aging BrainCollege of Physicians and SurgeonsColumbia UniversityNew York New York USA

Funder

U.S. Public Health Service

Arizona Alzheimer’s Consortium

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.09-139634

Reference81 articles.

1. Activated Microglial Cells Are Colocalized With Perivascular Deposits of Amyloid-β Protein in Alzheimer’s Disease Brain

2. Phagocytic microglia release cytokines and cytotoxins that regulate the survival of astrocytes and neurons in culture

3. Inflammation, anti-inflammatory agents and Alzheimer disease: The last 12 years

4. Senile plaques stimulate microglia to release a neurotoxin found in Alzheimer brain

5. Role of inflammation in neurodegenerative diseases

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