Enhanced DNA binding capacity on up‐regulated epidermal wild‐type p53 in vitiligo by H 2 O 2 ‐mediated oxidation: a possible repair mechanism for DNA damage-Reference-Cited by-同舟云学术

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Enhanced DNA binding capacity on up‐regulated epidermal wild‐type p53 in vitiligo by H 2 O 2 ‐mediated oxidation: a possible repair mechanism for DNA damage

Published:2009-07-29 Issue:11 Volume:23 Page:3790-3807
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Salem Mohamed M. A. E. L.¹,Shalbaf Mohammad¹,Gibbons Nicholas C. J.¹²,Chavan Bhaven¹,Thornton J. M.¹,Schallreuter Karin U.¹³

Affiliation:

1. Clinical and Experimental Dermatology Department of Biomedical Sciences/Centre for Skin Sciences School of Life Sciences University of Bradford Bradford UK

2. University of Bradford Bradford UK

3. Institute for Pigmentary Disorders in association with E. M. Arndt University Greifswald Germany

Funder

University of Bradford

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.09-132621

Reference115 articles.

1. Presence of epidermal allantoin further supports oxidative stress in vitiligo

2. Vitiligo pathogenesis: autoimmune disease, genetic defect, excessive reactive oxygen species, calcium imbalance, or what else?

3. In Vivo and In Vitro Evidence for Hydrogen Peroxide (H2O2) Accumulation in the Epidermis of Patients with Vitiligo and its Successful Removal by a UVB-Activated Pseudocatalase

4. Perturbed 6-Tetrahydrobiopterin Recycling via Decreased Dihydropteridine Reductase in Vitiligo: More Evidence for H2O2 Stress

5. Functional and structural index ofcellular impairment during vitiligo;Dell'Anna M. L.;J. Invest. Dermatol.,2006

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