The matricellular protein CCN1 enhances TGF‐β1/SMAD3‐dependent profibrotic signaling in fibroblasts and contributes to fibrogenic responses to lung injury-Reference-Cited by-同舟云学术

The matricellular protein CCN1 enhances TGF‐β1/SMAD3‐dependent profibrotic signaling in fibroblasts and contributes to fibrogenic responses to lung injury

Published:2016-02-16 Issue:6 Volume:30 Page:2135-2150
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Kurundkar Ashish R.¹,Kurundkar Deepali¹,Rangarajan Sunad¹,Locy Morgan L.¹,Zhou Yong¹,Liu Rui‐Ming¹,Zmijewski Jaroslaw¹,Thannickal Victor J.

Affiliation:

1. Division of Pulmonary, Allergy, and Critical Care MedicineDepartment of MedicineUniversity of Alabama at BirminghamBirminghamAlabamaUSA

Funder

National Institutes of Health

National Heart, Lung, and Blood Institute

National Institute on Aging

U.S. Department of Veterans Affairs

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201500173

Reference57 articles.

1. Matricellular proteins: extracellular modulators of cell function

2. Revisiting the matricellular concept

3. The de-adhesive activity of matricellular proteins: is intermediate cell adhesion an adaptive state?

4. Matricellular proteins as modulators of wound healing and the foreign body response

5. CCN family proteins and angiogenesis: from embryo to adulthood

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