IL‐17A promotes susceptibility during experimental visceral leishmaniasis caused by Leishmania donovani
Author:
Affiliation:
1. Department of PathologyThe Ohio State UniversityColumbusOhioUSA
2. Department of Veterinary BiosciencesThe Ohio State UniversityColumbusOhioUSA
3. Department of Internal MedicineThe Ohio State UniversityColumbusOhioUSA
Funder
National Institutes of Health
National Institute of Allergy and Infectious Diseases
Publisher
Wiley
Subject
Genetics,Molecular Biology,Biochemistry,Biotechnology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.15-277202
Reference47 articles.
1. Leishmaniasis: clinical syndromes and treatment
2. Macrophage Microbicidal Mechanisms In Vivo: Reactive Nitrogen versus Oxygen Intermediates in the Killing of Intracellular Visceral Leishmania donovani
3. STAT 4 is critical for immunity but not for antileishmanial activity of antimonials in experimental visceral leishmaniasis
4. Comparison of the A2 Gene Locus in Leishmania donovani and Leishmania major and Its Control over Cutaneous Infection
5. Leishmania major infection in interleukin-4 and interferon-gamma depleted mice
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