Inhibition of the HEG1–KRIT1 interaction increases KLF4 and KLF2 expression in endothelial cells-Reference-Cited by-同舟云学术

Inhibition of the HEG1–KRIT1 interaction increases KLF4 and KLF2 expression in endothelial cells

Published:2021-02-18 Issue:5 Volume:3 Page:334-355
ISSN:2573-9832
Container-title:FASEB BioAdvances
language:en
Short-container-title:FASEB BioAdvances

Author:

Lopez‐Ramirez Miguel Alejandro¹²,McCurdy Sara¹,Li Wenqing¹,Haynes Mark K.³,Hale Preston¹,Francisco Karol⁴⁵,Oukoloff Killian⁵,Bautista Matthew¹,Choi Chelsea H.J.¹,Sun Hao¹,Gongol Brendan¹^ORCID,Shyy John Y.¹,Ballatore Carlo⁵,Sklar Larry A.³,Gingras Alexandre R.¹^ORCID

Affiliation:

1. Department of Medicine University of California San Diego La Jolla CA USA

2. Department of Pharmacology University of California San Diego La Jolla CA USA

3. Department of Pathology Center for Molecular DiscoveryUniversity of New Mexico School of Medicine Albuquerque NM USA

4. Department of Chemistry & Biochemistry University of California San Diego La Jolla CA USA

5. Skaggs School of Pharmacy and Pharmaceutical Sciences University of California San Diego La Jolla CA USA

Funder

National Institutes of Health

Publisher

Wiley

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fba.2020-00141

Reference50 articles.

1. Regulation of cardiovascular development and integrity by the heart of glass–cerebral cavernous malformation protein pathway

2. Structural basis of the junctional anchorage of the cerebral cavernous malformations complex

3. The Structure of the Ternary Complex of Krev Interaction Trapped 1 (KRIT1) Bound to Both the Rap1 GTPase and the Heart of Glass (HEG1) Cytoplasmic Tail

4. KRIT-1/CCM1 is a Rap1 effector that regulates endothelial cell–cell junctions

5. Structural basis of the junctional anchorage of the cerebral cavernous malformations complex

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