Transgene overexpression of αB crystallin confers simultaneous protection against cardiomyocyte apoptosis and necrosis during myocardial ischemia and reperfusion
Author:
Affiliation:
1. Cardiovascular Research CenterDepartment of SurgeryUniversity of Connecticut Health CenterFarmington Connecticut 06030‐1110USA
2. Department of MedicineUniversity of California at San DiegoSan DiegoCaliforniaUSA
Publisher
Wiley
Subject
Genetics,Molecular Biology,Biochemistry,Biotechnology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.00-0199com
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2. [60] Antioxidant effectiveness in ischemia-reperfusion tissue injury
3. Phospholipid alterations in canine ischemic myocardium. Temporal and topographical correlations with Tc-99m-PPi accumulation and an in vitro sarcolemmal Ca2+ permeability defect.
4. Apoptotic and necrotic myocyte cell deaths are independent contributing variables ofinfarct size in rats;Kajstura J.;Lab. Invest.,1996
5. Oxidative Stress Developed During the Reperfusion of Ischemic Myocardium Induces Apoptosis
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