Neuronal activity‐induced SUMOylation of Akt1 by PIAS3 is required for long‐term potentiation of synaptic transmission-Reference-Cited by-同舟云学术

Neuronal activity‐induced SUMOylation of Akt1 by PIAS3 is required for long‐term potentiation of synaptic transmission

Published:2021-07-19 Issue:8 Volume:35 Page:
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Meng Li¹,Du Cai‐Ping¹,Lu Chun‐Yuan¹,Zhang Kun¹,Li Lin¹,Yan Jing‐Zhi¹,Hou Xiao‐Yu¹²^ORCID

Affiliation:

1. Jiangsu Key Laboratory of Brain Disease Bioinformation Research Center for Biochemistry and Molecular Biology Xuzhou Medical University Xuzhou China

2. State Key Laboratory of Natural Medicines School of Life Science and Technology China Pharmaceutical University Nanjing China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.202002728R

Reference50 articles.

1. Synaptic plasticity of NMDA receptors: mechanisms and functional implications

2. Long-term potentiation and the role of N -methyl- d -aspartate receptors

3. SUMOylation regulates kainate-receptor-mediated synaptic transmission

4. Activity-dependent SUMOylation of the brain-specific scaffolding protein GISP

5. Novel role and mechanism of protein inhibitor of activated STAT1 in spatial learning

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