Skin Diseases and Tryptophan Metabolism
Author:
Affiliation:
1. Department of Dermatology, Asahi University Hospital
2. Department of Disease Control and Prevention, Fujita Health University Graduate School of Health Sciences
Publisher
The Society of Japanese Women Scientists
Subject
General Medicine
Link
https://www.jstage.jst.go.jp/article/sjws/23/0/23_230003/_pdf
Reference34 articles.
1. 1) Fernández-Gallego N, Sánchez-Madrid F, Cibrian D. Role of AHR ligands in skin homeostasis and cutaneous inflammation. Cells. 2021; 10: 3176.
2. 2) 斉藤邦明，藤垣未和子，清島 満．トリプトファン代謝と疾患―生体防御と破壊．臨床化学．2000; 29: 103–112.
3. 3) Badawy AA, Guillemin G. The plasma [kynurenine] / [tryptophan] ratio and indoleamine 2,3-dioxygenase: Time for appraisal. Int J Tryptophan Res. 2019; 12: 1178646919868978.
4. 4) Swimm A, Giver CR, DeFilipp Z, et al. Indoles derived from intestinal microbiota act via type I interferon signaling to limit graft-versus-host disease. Blood. 2018; 132: 2506–2519.
5. 5) Su X, Gao Y, Yang R. Gut microbiota-derived tryptophan metabolites maintain gut and systemic homeostasis. Cells. 2022; 11: 2296.
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