Redox Regulation of Cell Survival by the Thioredoxin Superfamily: An Implication of Redox Gene Therapy in the Heart-Reference-Cited by-同舟云学术

Redox Regulation of Cell Survival by the Thioredoxin Superfamily: An Implication of Redox Gene Therapy in the Heart

Published:2009-11 Issue:11 Volume:11 Page:2741-2758
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Ahsan Md. Kaimul¹,Lekli Istvan¹,Ray Diptarka¹,Yodoi Junji²,Das Dipak K.¹

Affiliation:

1. Cardiovascular Research Center, Department of Surgery, School of Medicine, University of Connecticut Health Center, Farmington, Connecticut.

2. Department of Biological Responses, Institute for Virus Research, Kyoto University, Kawahara-cho, Sakyo-ku, Kyoto, Japan.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2009.2683

Reference209 articles.

1. S-Glutathiolation of Ras Mediates Redox-sensitive Signaling by Angiotensin II in Vascular Smooth Muscle Cells

2. A Redox-Dependent Pathway for Regulating Class II HDACs and Cardiac Hypertrophy

3. Thioredoxin1 as a Negative Regulator of Cardiac Hypertrophy

4. Thioredoxin1 Upregulates Mitochondrial Proteins Related to Oxidative Phosphorylation and TCA Cycle in the Heart

5. Antioxidant Function of the Mitochondrial Protein SP-22 in the Cardiovascular System

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