ROS-Dependent Signaling Mechanisms for Hypoxic Ca2+Responses in Pulmonary Artery Myocytes-Reference-Cited by-同舟云学术

ROS-Dependent Signaling Mechanisms for Hypoxic Ca2+Responses in Pulmonary Artery Myocytes

Published:2010-03 Issue:5 Volume:12 Page:611-623
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Wang Yong-Xiao¹,Zheng Yun-Min¹

Affiliation:

1. Center for Cardiovascular Sciences, Albany Medical College, Albany, New York.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2009.2877

Reference122 articles.

1. Protein Kinase Cα Phosphorylates the TRPC1 Channel and Regulates Store-operated Ca2+ Entry in Endothelial Cells

2. Activation of store‐operated channels by noradrenaline via protein kinase C in rabbit portal vein myocytes

3. Mitochondrial metabolism, redox signaling, and fusion: a mitochondria-ROS-HIF-1α-Kv1.5 O2-sensing pathway at the intersection of pulmonary hypertension and cancer

4. A redox-based O2 sensor in rat pulmonary vasculature.

5. Impairment of hypoxic pulmonary vasoconstriction in mice lacking the voltage‐gated potassium channel Kv1.5

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