Defective Function of CD24+CD38+ Regulatory B Cells in Ankylosing Spondylitis
Author:
Affiliation:
1. Department of Orthopedics, Shandong Qianfoshan Hospital, Shandong University, Jinan, China.
2. BGC Biotechnology Research Center, Jinan, China.
3. Department of Orthopedics, The Second Hospital of Shandong University, Jinan, China.
Publisher
Mary Ann Liebert Inc
Subject
Cell Biology,Genetics,Molecular Biology,General Medicine
Link
http://www.liebertpub.com/doi/pdf/10.1089/dna.2015.3046
Reference36 articles.
1. Anti-interleukin-17A monoclonal antibody secukinumab in treatment of ankylosing spondylitis: a randomised, double-blind, placebo-controlled trial
2. CD19+CD24hiCD38hi B Cells Exhibit Regulatory Capacity in Healthy Individuals but Are Functionally Impaired in Systemic Lupus Erythematosus Patients
3. Th17 Cells Expressing KIR3DL2+ and Responsive to HLA-B27 Homodimers Are Increased in Ankylosing Spondylitis
4. Ankylosing spondylitis
5. Susceptibility to ankylosing spondylitis in twins the role of genes, HLA, and the environment
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