Overexpression of Human Thioredoxin in Transgenic Mice Controls Oxidative Stress and Life Span-Reference-Cited by-同舟云学术

Overexpression of Human Thioredoxin in Transgenic Mice Controls Oxidative Stress and Life Span

Published:2002-08 Issue:4 Volume:4 Page:693-696
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Mitsui Akira¹,Hamuro Junji¹,Nakamura Hajime²,Kondo Norihiko²,Hirabayashi Yoko³,Ishizaki-Koizumi Sonoko¹,Hirakawa Tadashi¹,Inoue Tohru³,Yodoi Junji²

Affiliation:

1. Pharmaceutical Research Laboratories, Ajinomoto Co., Inc., 1-1 Suzuki-cho, Kawasaki, 210-8681, Japan

2. Department of Biological Responses, Institute for Virus Research, Kyoto University, 53 Shogoin, Kawara-cho, Sakyo-ku, Kyoto, 606-8507, Japan

3. Cellular and Molecular Toxicology Division, Biological Safety Research Center, National Institute of Health Sciences, 1-18-1 Kamiyoga, Setagaya-ku, Tokyo, 158-8501, Japan

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/15230860260220201

Reference18 articles.

1. Disease states associated with telomerase deficiency appear earlier in mice with short telomeres

2. THIOREDOXIN

3. Hemin-induced Activation of the Thioredoxin Gene by Nrf2

4. The p66shc adaptor protein controls oxidative stress response and life span in mammals

5. Reactive oxygen-reducing and protein-refolding activities of adult t cell leukemia-derived factor /human thioredoxin

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