STAT3-Activating Cytokines: A Therapeutic Opportunity for Inflammatory Bowel Disease?
Author:
Affiliation:
1. The Walter and Eliza Hall Institute of Medical Research, Melbourne, Australia.
2. Department of Medical Biology, University of Melbourne, Melbourne, Australia.
Publisher
Mary Ann Liebert Inc
Subject
Virology,Cell Biology,Immunology
Link
http://www.liebertpub.com/doi/pdf/10.1089/jir.2014.0225
Reference121 articles.
1. Induced somatic inactivation of STAT3 in mice triggers the development of a fulminant form of enterocolitis
2. Investigation of Crohn's Disease Risk Loci in Ulcerative Colitis Further Defines Their Molecular Relationship
3. Interleukin-22, a Member of the IL-10 Subfamily, Induces Inflammatory Responses in Colonic Subepithelial Myofibroblasts
4. Blockade of interleukin 6 trans signaling suppresses T-cell resistance against apoptosis in chronic intestinal inflammation: Evidence in Crohn disease and experimental colitis in vivo
5. IL-22 mediates mucosal host defense against Gram-negative bacterial pneumonia
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