Receptor for Advanced Glycation End Products-Mediated Signaling Impairs the Maintenance of Bone Marrow Mesenchymal Stromal Cells in Diabetic Model Mice-Reference-Cited by-同舟云学术

Receptor for Advanced Glycation End Products-Mediated Signaling Impairs the Maintenance of Bone Marrow Mesenchymal Stromal Cells in Diabetic Model Mice

Published:2016-11-15 Issue:22 Volume:25 Page:1721-1732
ISSN:1547-3287
Container-title:Stem Cells and Development
language:en
Short-container-title:Stem Cells and Development

Author:

Aikawa Eriko¹,Fujita Ryo¹²³,Asai Maiko⁴,Kaneda Yasufumi³,Tamai Katsuto¹

Affiliation:

1. Department of Stem Cell Therapy Science, Graduate School of Medicine, Osaka University, Suita, Japan.

2. Department of Stem Cell Biology, Atomic Bomb Disease Institute, Nagasaki University Graduate School of Biomedical Sciences, Nagasaki, Japan.

3. Division of Gene Therapy Science, Graduate School of Medicine, Osaka University, Suita, Japan.

4. Faculty of Medicine, Hiroshima University, Higashihiroshima, Japan.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Developmental Biology,Hematology

Link

https://www.liebertpub.com/doi/pdf/10.1089/scd.2016.0067

Reference50 articles.

1. Multilineage Potential of Adult Human Mesenchymal Stem Cells

2. Marrow Stromal Cells as Stem Cells for Nonhematopoietic Tissues

3. Anti-inflammatory protein TSG-6 secreted by activated MSCs attenuates zymosan-induced mouse peritonitis by decreasing TLR2/NF-κB signaling in resident macrophages

4. Bone marrow stromal cells attenuate sepsis via prostaglandin E2–dependent reprogramming of host macrophages to increase their interleukin-10 production

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