Protein Transnitrosylation Signaling Networks Contribute to Inflammaging and Neurodegenerative Disorders-Reference-Cited by-同舟云学术

Protein Transnitrosylation Signaling Networks Contribute to Inflammaging and Neurodegenerative Disorders

Published:2021-09-01 Issue:7 Volume:35 Page:531-550
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Nakamura Tomohiro¹^ORCID,Oh Chang-ki¹,Zhang Xu¹,Tannenbaum Steven R.²,Lipton Stuart A.¹³^ORCID

Affiliation:

1. Department of Molecular Medicine and Neurodegeneration New Medicines Center, The Scripps Research Institute, La Jolla, California, USA.

2. Department of Biological Engineering, Massachusetts Institute of Technology, Cambridge, Massachusetts, USA.

3. Department of Neurosciences, University of California San Diego School of Medicine, La Jolla, California, USA.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

https://www.liebertpub.com/doi/pdf/10.1089/ars.2021.0081

Reference191 articles.

1. cGMP and S-nitrosylation: two routes for modulation of neuronal excitability by NO

2. OPA1, encoding a dynamin-related GTPase, is mutated in autosomal dominant optic atrophy linked to chromosome 3q28

3. DJ-1 protects the nigrostriatal axis from the neurotoxin MPTP by modulation of the AKT pathway

4. Shank3 mutation in a mouse model of autism leads to changes in the S-nitroso-proteome and affects key proteins involved in vesicle release and synaptic function

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