Hypothalamic Apelin/Reactive Oxygen Species Signaling Controls Hepatic Glucose Metabolism in the Onset of Diabetes-Reference-Cited by-同舟云学术

Hypothalamic Apelin/Reactive Oxygen Species Signaling Controls Hepatic Glucose Metabolism in the Onset of Diabetes

Published:2014-02 Issue:4 Volume:20 Page:557-573
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Drougard Anne¹²,Duparc Thibaut¹²,Brenachot Xavier¹²³,Carneiro Lionel³,Gouazé Alexandra³,Fournel Audren¹²,Geurts Lucie⁴,Cadoudal Thomas¹²,Prats Anne-Catherine⁵,Pénicaud Luc³,Vieau Didier⁶,Lesage Jean⁶,Leloup Corinne³,Benani Alexandre³,Cani Patrice D.⁴,Valet Philippe¹²,Knauf Claude¹²

Affiliation:

1. Institut National de la Santé et de la Recherche Médicale (INSERM), Toulouse, France.

2. Institut des Maladies Métaboliques et Cardiovasculaires (I2MC), Université Paul Sabatier, UPS, Toulouse Cedex, France.

3. UMR CNRS 6265-INRA 1324, Centre des Sciences du Goût et de l'Alimentation CSGA, Université de Bourgogne, Dijon, France.

4. Metabolism and Nutrition Research Group, Louvain Drug Research Institute, LDRI, Université Catholique de Louvain, Brussels, Belgium.

5. Université de Toulouse, UPS, TRADGENE, Toulouse, France.

6. Unité Environnement Périnatal et Croissance, EA 4489, IFR 114, Université Lille-Nord de France, Equipe Dénutritions Maternelles Périnatales, Villeneuve d'Ascq Cédex, France.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2013.5182

Reference58 articles.

1. New hepatic targets for glycaemic control in diabetes

2. Mitochondrial H2O2 emission and cellular redox state link excess fat intake to insulin resistance in both rodents and humans

3. Mitochondria Are the Source of Hydrogen Peroxide for Dynamic Brain-Cell Signaling

4. The p53 isoform, Δ133p53α, stimulates angiogenesis and tumour progression

5. Pancreatic α-cell dysfunction in diabetes

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