Ero1α Regulates Ca2+Fluxes at the Endoplasmic Reticulum–Mitochondria Interface (MAM)-Reference-Cited by-同舟云学术

Ero1α Regulates Ca2+Fluxes at the Endoplasmic Reticulum–Mitochondria Interface (MAM)

Published:2012-05-15 Issue:10 Volume:16 Page:1077-1087
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Anelli Tiziana¹²,Bergamelli Leda¹²,Margittai Eva²,Rimessi Alessandro³,Fagioli Claudio²,Malgaroli Antonio¹,Pinton Paolo³,Ripamonti Maddalena¹,Rizzuto Rosario³,Sitia Roberto¹²

Affiliation:

1. Università Vita-Salute San Raffaele, Milano, Italy.

2. Division of Genetics and Cell Biology, San Raffaele Scientific Institute, Milano, Italy.

3. Department of Experimental and Diagnostic Medicine, Section of General Pathology, Interdisciplinary Center for the Study of Inflammation (ICSI), BioPharmaNet, University of Ferrara, Ferrara, Italy.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2011.4004

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3. Sequential steps and checkpoints in the early exocytic compartment during secretory IgM biogenesis

4. Two Conserved Cysteine Triads in Human Ero1α Cooperate for Efficient Disulfide Bond Formation in the Endoplasmic Reticulum

5. B- to Plasma-Cell Terminal Differentiation Entails Oxidative Stress and Profound Reshaping of the Antioxidant Responses

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