The Role of Dynamin in HIV Type 1 Env-Mediated Cell–Cell Fusion
Author:
Affiliation:
1. Department of Surgery, Duke University Medical Center, Durham, North Carolina.
Publisher
Mary Ann Liebert Inc
Subject
Infectious Diseases,Virology,Immunology
Link
http://www.liebertpub.com/doi/pdf/10.1089/aid.2010.0259
Reference38 articles.
1. Apoptosis Control in Syncytia Induced by the HIV Type 1–Envelope Glycoprotein Complex
2. Mitochondrial Apoptosis Induced by the HIV-1 Envelope
3. Mechanisms of apoptosis induction by the HIV-1 envelope
4. Cell-to-cell transmission of human immunodeficiency virus type 1 in the presence of azidothymidine and neutralizing antibody
5. Cell-to-cell spread of HIV-1 occurs within minutes and may not involve the participation of virus particles
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1. Evidence Showing that Tetraspanins Inhibit HIV-1-Induced Cell-Cell Fusion at a Post-Hemifusion Stage;Viruses;2014-03-07
2. HIV entry: a game of hide-and-fuse?;Current Opinion in Virology;2014-02
3. Dynamin-2 Regulates Fusion Pore Expansion and Quantal Release through a Mechanism that Involves Actin Dynamics in Neuroendocrine Chromaffin Cells;PLoS ONE;2013-08-05
4. HIV Entry and Envelope Glycoprotein-mediated Fusion;Journal of Biological Chemistry;2012-11
5. Intracellular curvature-generating proteins in cell-to-cell fusion;Biochemical Journal;2011-11-14
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