A Reduction in Intracellular Reactive Oxygen Species Due to a Mutation in NCF4 Promotes Autoimmune Arthritis in Mice-Reference-Cited by-同舟云学术

A Reduction in Intracellular Reactive Oxygen Species Due to a Mutation in NCF4 Promotes Autoimmune Arthritis in Mice

Published:2016-12-20 Issue:18 Volume:25 Page:983-996
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Winter Susann¹,Hultqvist Hopkins Malin¹,Laulund Frida¹,Holmdahl Rikard¹

Affiliation:

1. Division of Medical Inflammation Research, Department of Medical Biochemistry and Biophysics, Karolinska Institute, Stockholm, Sweden.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2016.6675

Reference58 articles.

1. PtdIns3P and Rac direct the assembly of the NADPH oxidase on a novel, pre-phagosomal compartment during FcR-mediated phagocytosis in primary mouse neutrophils

2. Analysis of type II collagen-reactive T cells in the mouse I. Different regulation of autoreactivevs. non-autoreactive anti-type II collagen T cells in the DBA/1 mouse

3. Activation of neutrophil respiratory burst by fungal particles requires phosphatidylinositol 3-phosphate binding to p40phox in humans but not in mice

4. p40 Is Phosphorylated on Threonine 154 and Serine 315 during Activation of the Phagocyte NADPH Oxidase

5. The Crystal Structure of the PX Domain from p40phox Bound to Phosphatidylinositol 3-Phosphate

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