MyD88-Dependent Silencing of Transgene Expression During the Innate and Adaptive Immune Response to Helper-Dependent Adenovirus
Author:
Affiliation:
1. Department of Human and Molecular Genetics, Baylor College of Medicine, Houston, TX 77030.
2. University of Helsinki and Helsinki University Central Hospital, Helsinki 00290, Finland.
3. Howard Hughes Medical Institute, Chevy Chase, MD 20815.
Publisher
Mary Ann Liebert Inc
Subject
Genetics,Molecular Biology,Molecular Medicine
Link
http://www.liebertpub.com/doi/pdf/10.1089/hum.2009.155
Reference45 articles.
1. Targeted Disruption of the MyD88 Gene Results in Loss of IL-1- and IL-18-Mediated Function
2. Pathogen Recognition and Innate Immunity
3. Adenovirus Vector-Induced Innate Inflammatory Mediators, MAPK Signaling, As Well As Adaptive Immune Responses Are Dependent upon Both TLR2 and TLR9 In Vivo
4. One-year Expression From High-capacity Adenoviral Vectors in the Brains of Animals With Pre-existing Anti-adenoviral Immunity: Clinical Implications
5. Adenovirus efficiently transduces plasmacytoid dendritic cells resulting in TLR9-dependent maturation and IFN-α production
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