Defective Galactosylation and Clearance of IgA1 Molecules as a Possible Etiopathogenic Factor in IgA Nephropathy1-Reference-Cited by-同舟云学术

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Defective Galactosylation and Clearance of IgA1 Molecules as a Possible Etiopathogenic Factor in IgA Nephropathy1

Published: Issue: Volume: Page:172-182
ISSN:0302-5144
Container-title:Contributions to Nephrology
language:
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Author:

Mestecky Jiri,Tomana Milan,Crowley-Nowick Peggy A.,Moldoveanu Zina,Julian Bruce A.,Jackson Susan

Publisher

S. Karger AG

Cited by 167 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. B-Cell Epigenetic Modulation of IgA Response by 5-Azacytidine and IgA Nephropathy;Journal of the American Society of Nephrology;2024-08-13

2. Sparsentan ameliorates glomerular hypercellularity and inflammatory-gene networks induced by IgA1-IgG immune complexes in a mouse model of IgA nephropathy;American Journal of Physiology-Renal Physiology;2024-05-01

3. Non-immunosuppressive treatment for IgA nephropathy;Cochrane Database of Systematic Reviews;2024-02-01

4. The significance of galactose-deficient immunoglobulin A1 staining in kidney diseases with IgA deposition;International Urology and Nephrology;2023-02-27

5. IgA Nephropathy: Pleiotropic impact of Epstein-Barr virus infection on immunopathogenesis and racial incidence of the disease;Frontiers in Immunology;2023-02-07

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