BMI1 Inhibition Improves Lesion Burden in Cerebral Cavernous Malformations-Reference-Cited by-同舟云学术

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BMI1 Inhibition Improves Lesion Burden in Cerebral Cavernous Malformations

Published:2024-08-27 Issue:9 Volume:150 Page:738-741
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Valentino Mariaelena¹^ORCID,Malinverno Matteo¹,Maderna Claudio¹,Pham Van-Cuong²,Rödel Claudia Jasmin²^ORCID,Zanardi Federica¹,Arce Maximiliano³^ORCID,Drufuca Lorenzo⁴^ORCID,Rossetti Grazisa¹^ORCID,Magnusson Peetra U.³^ORCID,Lampugnani Maria Grazia¹^ORCID,Dejana Elisabetta¹^ORCID,Abdelilah-Seyfried Salim²,Pagani Massimiliano¹⁴^ORCID

Affiliation:

1. IFOM ETS, AIRC Institute of Molecular Oncology, Milan, Italy (M.V., M.M., C.M., F.Z., G.R., M.G.L., E.D., M.P.).

2. Institute of Biochemistry and Biology, Potsdam University, Germany (V.-C.P., C.J.R., S.A.-S.).

3. Department of Immunology, Genetics and Pathology, Uppsala University, Sweden (M.A., P.U.M.).

4. Department of Medical Biotechnology and Translational Medicine, Università degli Studi di Milano, Italy (L.D., M.P.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Reference5 articles.

1. Blocking Signalopathic Events to Treat Cerebral Cavernous Malformations

2. Immunothrombosis and vascular heterogeneity in cerebral cavernous malformation

3. Endothelial cell clonal expansion in the development of cerebral cavernous malformations

4. Bmi1 regulates self-renewal and epithelial to mesenchymal transition in breast cancer cells through Nanog

5. Self-renewal as a therapeutic target in human colorectal cancer

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