CSL112 (Apolipoprotein A‐I [Human]) Reduces the Elevation in Neutrophil‐to‐Lymphocyte Ratio Induced by Acute Myocardial Infarction
Author:
Affiliation:
1. CSL Ltd Bio21 Institute Melbourne Victoria Australia
2. CSL Behring King of Prussia PA USA
3. PERFUSE Study Group Boston and Beth Israel Deaconess Medical Center Harvard Medical School Boston MA USA
Publisher
Ovid Technologies (Wolters Kluwer Health)
Reference5 articles.
1. Early and late recurrent cardiovascular events among high‐risk patients with an acute coronary syndrome: Meta‐analysis of phase III studies and implications on trial design
2. Neutrophils in Post-myocardial Infarction Inflammation: Damage vs. Resolution?
3. The neutrophil–lymphocyte ratio and incident atherosclerotic events: analyses from five contemporary randomized trials
4. The CSL112-2001 trial: Safety and tolerability of multiple doses of CSL112 (apolipoprotein A-I [human]), an intravenous formulation of plasma-derived apolipoprotein A-I, among subjects with moderate renal impairment after acute myocardial infarction
5. Regulation of Hematopoietic Stem and Progenitor Cell Mobilization by Cholesterol Efflux Pathways
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1. Anti-inflammatory mechanism of Apolipoprotein A-I;Frontiers in Immunology;2024-07-08
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