TNF-α Contributes to Endothelial Dysfunction by Upregulating Arginase in Ischemia/Reperfusion Injury-Reference-Cited by-同舟云学术

TNF-α Contributes to Endothelial Dysfunction by Upregulating Arginase in Ischemia/Reperfusion Injury

Published:2007-06 Issue:6 Volume:27 Page:1269-1275
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:ATVB

Author:

Gao Xue¹,Xu Xiangbin¹,Belmadani Souad¹,Park Yoonjung¹,Tang Zhonghua¹,Feldman Arthur M.¹,Chilian William M.¹,Zhang Cuihua¹

Affiliation:

1. From the Department of Veterinary Physiology & Pharmacology (X.G., X.X., Y.P., C.Z.), Texas A&M University, College Station; the Department of Medicine (Z.T., A.M.F.), Jefferson Medical College, Philadelphia, Pa; and the Department of Physiology (S.B., W.M.C.), LSU Health Sciences Center, New Orleans, La.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference22 articles.

1. Ischemia‐reperfusion selectively impairs nitric oxide‐ mediated dilation in coronary arterioles: counteracting role of arginase

2. Endothelial Nitric Oxide Synthase in Vascular Disease

3. Cytokine treatment increases arginine metabolism and uptake in bovine pulmonary arterial endothelial cells

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