Genetic and Pharmacologic Inhibition of the Neutrophil Elastase Inhibits Experimental Atherosclerosis-Reference-Cited by-同舟云学术

Genetic and Pharmacologic Inhibition of the Neutrophil Elastase Inhibits Experimental Atherosclerosis

Published:2018-02-20 Issue:4 Volume:7 Page:
ISSN:2047-9980
Container-title:Journal of the American Heart Association
language:en
Short-container-title:JAHA

Author:

Wen Guanmei¹²³,An Weiwei¹,Chen Jiangyong¹⁴,Maguire Eithne M.¹,Chen Qishan¹⁵,Yang Feng¹⁵,Pearce Stuart W. A.¹,Kyriakides Maria¹,Zhang Li⁵,Ye Shu¹⁶,Nourshargh Sussan⁷,Xiao Qingzhong¹²³

Affiliation:

1. Centre for Clinical Pharmacology, William Harvey Research Institute, Barts and The London School of Medicine and Dentistry, Queen Mary University of London, London, United Kingdom

2. Key Laboratory of Cardiovascular Diseases, The Second Affiliated Hospital, School of Basic Medical Sciences, Guangzhou Medical University, Guangzhou, Guangdong, China

3. Key Laboratory of Protein Modification and Degradation, School of Basic Medical Sciences, Guangzhou Medical University, Guangzhou, Guangdong, China

4. Department of Cardiothoracic Surgery, Yongchuan Hospital of Chongqing Medical University, Chongqing, China

5. Department of Cardiology, the First Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, Zhejiang, China

6. Department of Cardiovascular Sciences, University of Leicester, Leicester, United Kingdom

7. Centre for Microvascular Research, William Harvey Research Institute, Barts and The London School of Medicine and Dentistry, Queen Mary University of London, London, United Kingdom

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference72 articles.

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4. Polymorphonuclear Cells Isolated from Human Peripheral Blood Cleave Lipoprotein(a) and Apolipoprotein(a) at Multiple Interkringle Sites via the Enzyme Elastase

5. Modification of low density lipoproteins by polymorphonuclear cell elastase leads to enhanced uptake by human monocyte‐derived macrophages via the low density lipoprotein receptor pathway;Polacek D;J Lipid Res,1988

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