TRPC4 Knockout Mice
Author:
Affiliation:
1. From the Transmembrane Signaling Group, Laboratory of Signal Transduction, and Division of Intramural Research, National Institute of Environmental Health Sciences, Research Triangle Park, NC.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Cardiology and Cardiovascular Medicine,Physiology
Link
https://www.ahajournals.org/doi/pdf/10.1161/01.RES.0000026600.67226.92
Reference15 articles.
1. Impairment of Store-Operated Ca 2+ Entry in TRPC4 −/− Mice Interferes With Increase in Lung Microvascular Permeability
2. Lack of an endothelial store-operated Ca2+ current impairs agonist-dependent vasorelaxation in TRP4−/− mice
3. Thapsigargin, a novel molecular probe for studying intracellular calcium release and storage
4. Capacitative calcium entry revisited
5. Depletion of intracellular calcium stores activates a calcium current in mast cells
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1. Activation of calcium-sensing receptor increases TRPC3 expression in rat cardiomyocytes;Biochemical and Biophysical Research Communications;2011-03
2. Calcium/Calmodulin-dependent Protein Kinase II Delta 6 (CaMKIIδ6) and RhoA Involvement in Thrombin-induced Endothelial Barrier Dysfunction;Journal of Biological Chemistry;2010-07
3. Calcium-sensing receptor activation contributed to apoptosis stimulates TRPC6 channel in rat neonatal ventricular myocytes;Biochemical and Biophysical Research Communications;2010-04
4. Intracellular Ca2+ store depletion induces the formation of macromolecular complexes involving hTRPC1, hTRPC6, the type II IP3 receptor and SERCA3 in human platelets;Biochimica et Biophysica Acta (BBA) - Molecular Cell Research;2008-06
5. Regulation of Endothelial Junctional Permeability;Annals of the New York Academy of Sciences;2008-03-19
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