Endogenous RGS Proteins and Gα Subtypes Differentially Control Muscarinic and Adenosine-Mediated Chronotropic Effects-Reference-Cited by-同舟云学术

Endogenous RGS Proteins and Gα Subtypes Differentially Control Muscarinic and Adenosine-Mediated Chronotropic Effects

Published:2006-03-17 Issue:5 Volume:98 Page:659-666
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

Fu Ying¹,Huang Xinyan¹,Zhong Huailing¹,Mortensen Richard M.¹,D’Alecy Louis G.¹,Neubig Richard R.¹

Affiliation:

1. From the Departments of Pharmacology (Y.F., X.H., H.Z., R.M.M., R.R.N.), Molecular and Integrative Physiology (R.M.M., L.G.D’A.), and Surgery (Vascular) (L.G.D’A.), University of Michigan, Ann Arbor; and the Department of Surgery (L.G.D’A.), William Beaumont Hospital, Royal Oak, Mich.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference43 articles.

1. Role of G Proteins in the Regulation of the Cardiovascular System

2. Targeted inactivation of i2 or i3 disrupts activation of the cardiac muscarinic K+ channel, IK+Ach, in intact cells

3. G o is necessary for muscarinic regulation of Ca2+ channels in mouse heart

4. G αi2, G αi3and G αoare all Required for Normal Muscarinic Inhibition of the Cardiac Calcium Channels in Nodal/Atrial-like Cultured Cardiocytes

5. RGS8 accelerates G-protein-mediated modulation of K+currents

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