Norepinephrine-Induced Stimulation of Kir4.1/Kir5.1 Is Required for the Activation of NaCl Transporter in Distal Convoluted Tubule
Author:
Affiliation:
1. From the Department of Physiology, Harbin Medical University, China (X.-P.D., L.G., Y.X., Y.-H.Z., X.-X.M., J.-L.W., D.-D.Z., W.-H.W., R.G.)
2. Department of Pharmacology, New York Medical College, Valhalla (Z.-X.G., P.W., D.-H.L., W.-H.W.).
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Internal Medicine
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4. Chloride Sensing by WNK1 Involves Inhibition of Autophosphorylation
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2. Kir4.1 deletion prevents salt-sensitive hypertension in early streptozotocin-induced diabetic mice via Na+–Cl− cotransporter in the distal convoluted tubule;Journal of Hypertension;2023-04-04
3. Activation of Kir4.1/Kir5.1 contributes to the cyclosporin A‐induced stimulation of the renal NaCl cotransporter and hyperkalemic hypertension;Acta Physiologica;2023-02-16
4. Blood pressure effects of sodium transport along the distal nephron;Kidney International;2022-12
5. Inwardly rectifying K+ channels 4.1 and 5.1 (Kir4.1/Kir5.1) in the renal distal nephron;American Journal of Physiology-Cell Physiology;2022-08-01
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