To E or Not To E—How Do We Tell?
Author:
Affiliation:
1. From the Department of Medicine, University of California San Diego.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Physiology (medical),Cardiology and Cardiovascular Medicine
Link
https://www.ahajournals.org/doi/pdf/10.1161/01.CIR.98.25.2785
Reference20 articles.
1. Low Density Lipoprotein Oxidation and Its Pathobiological Significance
2. The oxidation hypothesis of atherosclerosis
3. Steinberg D Witztum JL. Lipoproteins lipoprotein oxidation and atherogenesis. In Chien KR ed. Molecular Basis of Cardiovascular Disease . Philadelphia Pa: W.B. Saunders Co. 1998:458–475.
4. Vitamin E suppresses isoprostane generation in vivo and reduces atherosclerosis in ApoE-deficient mice
5. A series of prostaglandin F2-like compounds are produced in vivo in humans by a non-cyclooxygenase, free radical-catalyzed mechanism.
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