Endothelin-1–Induced Arrhythmogenic Ca2+Signaling Is Abolished in Atrial Myocytes of Inositol-1,4,5-Trisphosphate(IP3)–Receptor Type 2

Endothelin-1–Induced Arrhythmogenic Ca2+Signaling Is Abolished in Atrial Myocytes of Inositol-1,4,5-Trisphosphate(IP3)–Receptor Type 2–Deficient Mice

Published:2005-06-24 Issue:12 Volume:96 Page:1274-1281
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

Li Xiaodong¹,Zima Aleksey V.¹,Sheikh Farah¹,Blatter Lothar A.¹,Chen Ju¹

Affiliation:

1. From the Department of Medicine (H.L., F.S., J.C.), University of California San Diego, La Jolla, Calif; and the Department of Physiology (A.V.Z., L.A.B.), Loyola University of Chicago, Stritch School of Medicine, Maywood, Ill.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

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