Impairment of Store-Operated Ca 2+ Entry in TRPC4 −/− Mice Interferes With Increase in Lung Microvascular Permeability-Reference-Cited by-同舟云学术

Impairment of Store-Operated Ca 2+ Entry in TRPC4 −/− Mice Interferes With Increase in Lung Microvascular Permeability

Published:2002-07-12 Issue:1 Volume:91 Page:70-76
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

Tiruppathi Chinnaswamy¹,Freichel Marc¹,Vogel Stephen M.¹,Paria Biman C.¹,Mehta Dolly¹,Flockerzi Veit¹,Malik Asrar B.¹

Affiliation:

1. From the Department of Pharmacology, The University of Illinois, Chicago, Ill; and Universitat des Saarlandes (M.F., V.F.), Institut fur Pharmakologie und Toxikologie, Homburg, Germany.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference21 articles.

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2. Thrombin-Mediated Increase in Vascular Endothelial Permeability

3. Abrogation of thrombin-induced increase in pulmonary microvascular permeability in PAR-1 knockout mice

4. Requirement for Ca2+ signaling in the mechanism of thrombin-induced increase in endothelial permeability

5. Ca 2+ signalling and PKCα activate increased endothelial permeability by disassembly of VE—cadherin junctions

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