A Dynamic Notch Injury Response Activates Epicardium and Contributes to Fibrosis Repair-Reference-Cited by-同舟云学术

A Dynamic Notch Injury Response Activates Epicardium and Contributes to Fibrosis Repair

Published:2011-01-07 Issue:1 Volume:108 Page:51-59
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circ Res

Author:

Russell Jamie L.¹,Goetsch Sean C.¹,Gaiano Nicholas R.¹,Hill Joseph A.¹,Olson Eric N.¹,Schneider Jay W.¹

Affiliation:

1. From the Departments of Internal Medicine/Cardiology (J.L.R., S.C.G., J.A.H., J.W.S.) and Molecular Biology (E.N.O.), University of Texas Southwestern Medical Center, Dallas; and Department of Neurology (N.R.G.), Johns Hopkins University, Baltimore, Md.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference30 articles.

1. The origin of fibroblasts and mechanism of cardiac fibrosis

2. Endothelial-to-mesenchymal transition contributes to cardiac fibrosis

3. Origin of Cardiac Fibroblasts and the Role of Periostin

4. Genetic fate mapping demonstrates contribution of epicardium-derived cells to the annulus fibrosis of the mammalian heart

5. Cardiac renewing: interstitial Cajal-like cells nurse cardiomyocyte progenitors in epicardial stem cell niches

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