Role of Asymmetric Dimethylarginine in Vascular Injury in Transgenic Mice Overexpressing Dimethylarginie Dimethylaminohydrolase 2-Reference-Cited by-同舟云学术

Role of Asymmetric Dimethylarginine in Vascular Injury in Transgenic Mice Overexpressing Dimethylarginie Dimethylaminohydrolase 2

Published:2007-07-20 Issue:2 Volume:101 Page:
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

Hasegawa Kazuhiro¹,Wakino Shu¹,Tatematsu Satoru¹,Yoshioka Kyoko¹,Homma Koichiro¹,Sugano Naoki¹,Kimoto Masumi¹,Hayashi Koichi¹,Itoh Hiroshi¹

Affiliation:

1. From the Department of Internal Medicine (K.H., S.W., S.T., K.Y., K.H., N.S., K.H., H.I.), Keio University, Tokyo, and the Department of Nutritional Science (M.K.), Okayama Prefectural University, Okayama, Japan.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference35 articles.

1. Accumulation of an endogenous inhibitor of nitric oxide synthesis in chronic renal failure

2. Plasma concentration of asymmetrical dimethylarginine and mortality in patients with end-stage renal disease: a prospective study

3. Identification of two human dimethylarginine dimethylaminohydrolases with distinct tissue distributions and homology with microbial arginine deiminases

4. Regulation of nitric oxide synthesis by dimethylarginine dimethylaminohydrolase

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