β-MyHC and Cardiac Hypertrophy
Author:
Affiliation:
1. From the Department of Pathology and Laboratory Medicine, University of North Carolina at Chapel Hill, Chapel Hill, NC.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Cardiology and Cardiovascular Medicine,Physiology
Link
https://www.ahajournals.org/doi/pdf/10.1161/CIRCRESAHA.111.252619
Reference9 articles.
1. Molecular Mechanisms of Myocardial Remodeling
2. Protooncogene induction and reprogramming of cardiac gene expression produced by pressure overload.
3. Fibrosis, not cell size, delineates beta-myosin heavy chain reexpression during cardiac hypertrophy and normal aging in vivo
4. Decreased beta-adrenergic responsiveness following hypertrophy occurs only in cardiomyocytes that also re-express beta-myosin heavy chain
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