Connexin 43 and CaV1.2 Ion Channel Trafficking in Healthy and Diseased Myocardium-Reference-Cited by-同舟云学术

Connexin 43 and CaV1.2 Ion Channel Trafficking in Healthy and Diseased Myocardium

Published:2016-06 Issue:6 Volume:9 Page:
ISSN:1941-3149
Container-title:Circulation: Arrhythmia and Electrophysiology
language:en
Short-container-title:Circ: Arrhythmia and Electrophysiology

Author:

Basheer Wassim A.¹,Shaw Robin M.¹

Affiliation:

1. From the Heart Institute and Department of Medicine, Cedars-Sinai Medical Center, Los Angeles, CA (W.A.B., R.M.S.); and Department of Medicine, University of California Los Angeles (R.M.S.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Link

https://www.ahajournals.org/doi/pdf/10.1161/CIRCEP.115.001357

Reference119 articles.

1. A 14-3-3 Mode-1 Binding Motif Initiates Gap Junction Internalization During Acute Cardiac Ischemia

2. Neurotrophin B receptor kinase increases Kv subfamily member 1.3 (Kv1.3) ion channel half-life and surface expression

3. Roles of a Membrane-localized βSubunit in the Formation and Targeting of Functional L-type Ca2+ Channels

4. Surface Traffic of Dendritic CaV1.2 Calcium Channels in Hippocampal Neurons

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