Role of JNK, p38, and ERK in Platelet-Derived Growth Factor–Induced Vascular Proliferation, Migration, and Gene Expression-Reference-Cited by-同舟云学术

Role of JNK, p38, and ERK in Platelet-Derived Growth Factor–Induced Vascular Proliferation, Migration, and Gene Expression

Published:2003-05 Issue:5 Volume:23 Page:795-801
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:ATVB

Author:

Zhan Yumei¹,Kim Shokei¹,Izumi Yasukatsu¹,Izumiya Yasuhiro¹,Nakao Takafumi¹,Miyazaki Hitoshi¹,Iwao Hiroshi¹

Affiliation:

1. From the Department of Pharmacology, Osaka City University Medical School, Osaka, Japan, and Gene Experiment Center and Center for Tsukuba Advanced Research Alliance (H.M.), University of Tsukuba, Ibaraki, Japan.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference38 articles.

1. The pathogenesis of atherosclerosis: a perspective for the 1990s

2. Platelet-derived growth factor promotes smooth muscle migration and intimal thickening in a rat model of balloon angioplasty.

3. Mechanism of Action and In Vivo Role of Platelet-Derived Growth Factor

4. Inhibition of Neointimal Smooth Muscle Accumulation After Angioplasty by an Antibody to PDGF

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