Thrombospondin-2 as a Potential Risk Factor in a General Population-Reference-Cited by-同舟云学术

Thrombospondin-2 as a Potential Risk Factor in a General Population

Published:2019-03-30 Issue:2 Volume:60 Page:310-317
ISSN:1349-2365
Container-title:International Heart Journal
language:en
Short-container-title:Int. Heart J.

Author:

Morikawa Nagisa¹,Adachi Hisashi¹²,Enomoto Mika¹,Fukami Ako¹,Kumagai Eita¹,Nakamura Sachiko¹,Nohara Yume¹,Nakao Erika¹,Kono Shoko¹,Tsuru Tomoko¹,Sakaue Akiko¹,Hamamura Hitoshi¹,Fukumoto Yoshihiro¹

Affiliation:

1. Division of Cardiovascular Medicine, Department of Internal Medicine, Kurume University School of Medicine Kurume

2. Department of Community Medicine, Kurume University School of Medicine

Publisher

International Heart Journal (Japanese Heart Journal)

Subject

Cardiology and Cardiovascular Medicine,General Medicine

Link

https://www.jstage.jst.go.jp/article/ihj/60/2/60_18-246/_pdf

Reference37 articles.

1. 1. Mustonen E, Ruskoaho H, Rysä J. Thrombospondins, potential drug targets for cardiovascular diseases. Basic Clin Pharmacol Toxicol 2013; 112: 4-12.

2. 2. Chatila K, Ren G, Xia Y, Huebener P, Bujak M, Frangogiannis NG. The role of the thrombospondins in healing myocardial infarcts. Cardiovasc Hematol Agents Med Chem 2007; 5: 21-7.

3. 3. Swinnen M, Vanhoutte D, Van Almen GC, et al. Absence of thrombospondin-2 causes age-related dilated cardiomyopathy. Circulation 2009; 120: 1585-97.

4. 4. Papageorgiou AP, Swinnen M, Vanhoutte D, et al. Thrombospondin-2 prevents cardiac injury and dysfunction in viral myocarditis through the activation of regulatory T-cells. Cardiovasc Res 2012; 94: 115-24.

5. 5. van Almen GC, Swinnen M, Carai P, et al. Absence of thrombospondin-2 increases cardiomyocyte damage and matrix disruption in doxorubicin-induced cardiomyopathy. J Mol Cell Cardiol 2011; 51: 318-28.

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