Postentry Events Are Responsible for Restriction of Productive Varicella-Zoster Virus Infection in Chinese Hamster Ovary Cells
Author:
Affiliation:
1. Department of Pediatrics, Infectious Diseases Section
2. Department of Microbiology, University of Colorado Health Sciences Center, Denver, Colorado 80262
Abstract
Publisher
American Society for Microbiology
Subject
Virology,Insect Science,Immunology,Microbiology
Link
https://journals.asm.org/doi/pdf/10.1128/JVI.00939-06
Reference70 articles.
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2. Sequential isolation of proteoglycan synthesis mutants by using herpes simplex virus as a selective agent: evidence for a proteoglycan-independent virus entry pathway
3. Banfield, B. W., Y. Leduc, L. Esford, R. J. Visalli, C. R. Brandt, and F. Tufaro. 1995. Evidence for an interaction of herpes simplex virus with chondroitin sulfate proteoglycans during infection. Virology208:531-539.
4. Bourdon-Wouters, C., M. P. Merville-Louis, C. Sadzot-Delvaux, P. Marc, J. Piette, P. Delree, G. Moonen, and B. Rentier. 1990. Acute and persistent varicella-zoster virus infection of human and murine neuroblastoma cell lines. J. Neurosci. Res.26:90-97.
5. Browne, H., B. Bruun, and T. Minson. 2001. Plasma membrane requirements for cell fusion induced by herpes simplex virus type 1 glycoproteins gB, gD, gH and gL. J. Gen. Virol.82:1419-1422.
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