Absence of a Red Blood Cell Phenotype in Mice with Hematopoietic Deficiency of SEC23B-Reference-Cited by-同舟云学术

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Absence of a Red Blood Cell Phenotype in Mice with Hematopoietic Deficiency of SEC23B

Published:2014-07-28 Issue:19 Volume:34 Page:3721-3734
ISSN:0270-7306
Container-title:Molecular and Cellular Biology
language:en
Short-container-title:Molecular and Cellular Biology

Author:

Khoriaty R.,Vasievich M. P.,Jones M.,Everett L.,Chase J.,Tao J.,Siemieniak D.,Zhang B.,Maillard I.,Ginsburg D.

Publisher

American Society for Microbiology

Subject

Cell Biology,Molecular Biology

Reference45 articles.

1. Congenital dyserythropoietic anemia type II: epidemiology, clinical appearance, and prognosis based on long-term observation;Heimpel;Blood,2003

2. The COPII pathway and hematologic disease;Khoriaty;Blood,2012

3. The cisternae decorating the red blood cell membrane in congenital dyserythropoietic anemia (type II) originate from the endoplasmic reticulum;Alloisio;Blood,1996

4. Mutations affecting the secretory COPII coat component SEC23B cause congenital dyserythropoietic anemia type II;Schwarz;Nat. Genet.,2009

5. COPII and the regulation of protein sorting in mammals;Zanetti;Nat. Cell Biol.,2012

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1. Cargo selection in endoplasmic reticulum–to–Golgi transport and relevant diseases;Journal of Clinical Investigation;2023-01-03

2. A common human missense mutation of vesicle coat protein SEC23B leads to growth restriction and chronic pancreatitis in mice;Journal of Biological Chemistry;2022-01

3. The congenital dyserythropoieitic anemias;Current Opinion in Hematology;2021-12-24

4. SEC23A rescues SEC23B-deficient congenital dyserythropoietic anemia type II;Science Advances;2021-11-26

5. Vesicular formation regulated by ERK/MAPK pathway mediates human erythroblast enucleation;Blood Advances;2021-11-17

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