Role of ICP0 in the Strategy of Conquest of the Host Cell by Herpes Simplex Virus 1
Author:
Affiliation:
1. The Marjorie B. Kovler Viral Oncology Laboratories, The University of Chicago, Chicago, Illinois 60637
Publisher
American Society for Microbiology
Subject
Virology,Insect Science,Immunology,Microbiology
Link
https://journals.asm.org/doi/pdf/10.1128/JVI.78.5.2169-2178.2004
Reference163 articles.
1. Characterization of herpes simplex virus 1 alpha proteins 0, 4, and 27 with monoclonal antibodies
2. The Disappearance of Cyclins A and B and the Increase in Activity of the G 2 /M-Phase Cellular Kinase cdc2 in Herpes Simplex Virus 1-Infected Cells Require Expression of the α22/U S 1.5 and U L 13 Viral Genes
3. Posttranslational Processing of Infected Cell Proteins 0 and 4 of Herpes Simplex Virus 1 Is Sequential and Reflects the Subcellular Compartment in Which the Proteins Localize
4. E2F Proteins Are Posttranslationally Modified Concomitantly with a Reduction in Nuclear Binding Activity in Cells Infected with Herpes Simplex Virus 1
5. Advani, S. J., R. R. Weichselbaum, and B. Roizman. 2000. The role of cdc2 in the expression of herpes simplex virus genes. Proc. Natl. Acad. Sci. USA97:10996-11001.
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