Author:
Ahlgren T,Florin I,Jarstrand C,Thelestam M
Abstract
Clostridium difficile cytotoxin caused an irreversible dose- and time-dependent loss of fibronectin from the surfaces of human lung fibroblasts, paralleling the appearance of the cytopathic effect. Fibronectin was not required for the intoxication process. The results lend further support to a transmembrane connective link between fibronectin and the microfilaments.
Publisher
American Society for Microbiology
Subject
Infectious Diseases,Immunology,Microbiology,Parasitology
Reference26 articles.
1. Effects of cytochalasin B and colchicine on attachment of a major surface protein of fibroblasts;Ali I. U.;Biochim. Biophys. Acta,1977
2. Clostridiuni difficile and antibiotic-associated diarrhoea;Aronsson B.;Sweden. Scand. J. Infect. Dis. Suppl.,1982
3. Two toxins (D-1 and D-2) of Clostridium difficile causing antibiotic-associated colitis: purification and some characterization;Banno Y.;Biochem. Int.,1981
4. Role of Clostridium difficile in antibiotic-associated pseudomembraneous colitis;Bartlett J. G.;Gastroenterology,1978
5. Clindamycin-induced enterocolitis in hamsters as a model of pseudomembranous colitis in patients;Chang T.;Infect. Immun.,1978
Cited by
12 articles.
订阅此论文施引文献
订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献