Nothing in Excess—Lessons Learned from the Expression of High-Mobility Group Proteins Type a in Non-Cancer and Cancer Cells
Author:
Publisher
Informa UK Limited
Subject
Biotechnology
Link
http://www.tandfonline.com/doi/pdf/10.5504/BBEQ.2011.0102
Reference45 articles.
1. Inhibition of Nucleotide Excision Repair by High Mobility Group Protein HMGA1
2. A novel human DNA glycosylase that removes oxidative DNA damage and is homologous to Escherichia coli endonuclease VIII
3. A mitogen- and anisomycin-stimulated kinase phosphorylates HMG-14 in its basic amino-terminal domain in vivo and on isolated mononucleosomes.
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