Experimental Petit Mal-Like Seizure Induced by Microinjection of Kainic Acid Into the Unilateral Mediodorsal Nucleus of the Thalamus
Author:
Affiliation:
1. Department of Neurosurgery, Tokyo Women’s Medical University
2. Department of Neurosurgery, Asahikawa Medical College
Publisher
Japan Neurosurgical Society
Subject
Neurology (clinical),Surgery
Link
http://www.jstage.jst.go.jp/article/nmc/48/7/48_7_285/_pdf
Reference28 articles.
1. Kainic acid-induced thalamic seizure in cats — a possible model of petit mal seizure
2. γ-Hydroxybutyric acid induced spike and wave discharges in rats: Relation to high-affinity [3H]γ-hydroxybutyric acid binding sites in the thalamus and cortex
3. Thalamic mediodorsal and intralaminar nuclear lesions disrupt the generation of experimentally induced generalized absence-like seizures in rats
4. Thalamic NMDA receptors in the γ-hydroxybutyrate model of absence seizures: A cerebral microinjection study in rats
5. A model of atypical absence seizures: EEG, pharmacology, and developmental characterization
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